A patient with lower extremity weakness after recent endovascular repair of an abdominal aortic aneurysm
Thrombosis of an endovascular aortic repair (EVAR) is a devastating complication of a common surgical procedure that can lead to serious morbidity and mortality if not promptly recognized. This is the first case report of an EVAR graft thrombosis in the emergency medicine literature.
Introduction - Shaggy aorta is related to early adverse events following endovascular aneurysm repair (EVAR) for abdominal aortic aneurysm, specially peripheral and visceral embolization from the aorta.
Postimplantation syndrome (PIS) is a systemic inflammatory response occurring in an early phase after abdominal aortic aneurysm (AAA) endovascular repair (EVAR). The pathophysiology underlying PIS is not yet well understood. It is speculated that the type of the stent graft or the mural thrombus within the AAA may play a role in determing this inflammatory response. At present, there is no consensus about the influence of PIS on clinical outcomes during follow-up. The endovascular aneurysm sealing (EVAS) with the Nellix sac-anchoring endoprosthesis (Nellix Endovascular, Palo Alto, Calif) is a novel modality for AAA repair ...
ConclusionThe endovascular neck stabilization is a useful treatment option that facilitates standard EVAR for AAA in chronic aortic dissection.
CONCLUSIONSSecond-stage endovascular repair after previous fET is feasible with good mid-term results. This staged hybrid procedure is extremely effective in patients whose aneurysms are confined both to the arch and thoraco-abdominal aorta leading to an excellent functional result. In case of favorable anatomy endovascular repair in residual type B aortic dissection lead to complete false lumen thrombosis.
We examined the relationship between preoperative spatial morphology of ILT and the incidence of postoperative T2E.
Rationale: Abdominal aortic aneurysm is an extremely rare cause of deep vein thrombosis. Here we report an elderly gentleman who presented with deep vein thrombosis and was found to have concomitant abdominal aortic aneurysm upon ultrasonographic screening. It illustrates the possibility of such an aetiology, and the importance of screening for such aneurysms in a select patient population before heparinization. Patient concerns: A 73-year-old Asian gentleman with underlying hypertension, hyperlipidaemia, chronic renal failure, and history of chronic smoking presented to the emergency department with acute left lower ...
An 81 year old male underwent endovascular aneurysm repair (EVAR) (Endurant II-Medtronic) for a ruptured abdominal aortic aneurysm. During follow up he presented with a ruptured left common iliac aneurysm (57 mm, previously 36 mm), the result of a type IB endoleak, which was treated endovascularly (Anaconda limb and left hypogastric artery embolisation). After six months, the patient underwent femorofemoral crossover bypass because of left graft limb occlusion as a result of acute thrombosis and acute lower limb ischaemia.
Testicular infarction is a rare but important complication of endovascular abdominal aortic aneurysm repair (EVAR). Six previously reported cases included acute and latent postoperative presentations. Etiologies included delayed thrombosis of the aneurysmal sac or thromboembolic events in the setting of poor collateral vascular supply to the ischemic or infarcted testicle (1). This is the seventh reported case of testicular infarction, most likely secondary to routine occlusion of a right testicular artery, which was the only end artery supplying the right testicle.
With the introduction of EVAR more than twenty years ago, patients with AAA were presented with a new option for a treatment wherein they left the hospital in days rather than weeks, and recovered back to their usual activities in weeks rather than months. However, with these new opportunities came new challenges, such as the concept of an intraluminally repaired aortic aneurysm as a “chronic disease” which requires surveillance, and potentially have complications from endoleaks, limb thrombosis, or delayed rupture requiring further testing and costly interventions. With these choices emerges an opportunity to ...
CONCLUSIONS: Higher preprocedural fibrinogen, absence of persistent type II endoleak and higher age were predictive factors of aneurysm sac regression post-EVAR. PMID: 30874486 [PubMed - as supplied by publisher]