Inhibiting sEH suppresses NF- κB p65 signaling and reduces CXCL10 expression as a potential therapeutic target in HT

CONCLUSIONS: Our findings suggest that sEH/NF-κB p65/CXCL10-CXCR3 might be promising therapeutic targets for HT.PMID:38478377 | DOI:10.1210/clinem/dgae163

Source: The Journal of Clinical Endocrinology and Metabolism - March 13, 2024 Category: Endocrinology Authors: Jing Feng Xianghong Xu Wei Cai Xingwen Yang Ruilan Niu Ziqi Han Limin Tian Source Type: research