Metabolic regulation by PPARγ is required for IL-33-mediated activation of ILC2s in lung and adipose tissue

This study demonstrates that the metabolic sensor PPARγ is central for ILC2s activation, proliferation and function in lung and adipose tissue.

Source: Mucosal Immunology - October 26, 2020 Category: Allergy & Immunology Authors: Tinhinane Fali Tegest Aychek Maroua Ferhat Jean-Yves Jouzeau Meinrad Busslinger David Moulin G érard Eberl Source Type: research