A strong union is our best defence
Strengthening and building our union is the best defence for our members and our public services after the devastating election defeat, UNISON general secretary Dave Prentis told the national executive council’s December meeting. The scale of the defeat was “devastating for our union, devastating for our members and devastating for our public services,” he said. “But the answers to why we lost are complex and deserve proper consideration rather than just our own views. “What we need to focus on now is how we strengthen the union to face the challenges we know we will now face. “We did it in 2010 when we knew we faced austerity – that’s when we introduced fighting fund organisers and changed the way our industrial action fund worked. “We will need to streamline to the issues where we will have to defend our members. We have to focus on what matters to our members. And above all else, we need to keep them with us.” A re-invigorated Conservative government was likely to mean renewed attacks on trade unions, attacks on individual rights, and a continued risk of a no-deal Brexit, he warned. And the trade deals that will now be negotiated will risk opening up “all of our public services, not just the NHS” to competitive tender alongside international private companies. So “today is not for sitting around theorising or casting blame – we need to be strong, as a party and as a union”. The NEC al...
This report continues the development of ALIs as a clinical tool in wildlife while systematically testing one possible method for determining an optimal ALI for a particular species. PMID: 32228119 [PubMed - as supplied by publisher]
Authors: Park J, Kim J, Chen Y, Song HC, Chen Y, Zheng M, Surh YJ, Kim UH, Park JW, Joe Y, Chung HT Abstract Oxidative stress is recognised as a key factor that can lead to cellular senescence and aging. Carbon monoxide (CO) is produced by haemoxygenase-1 (HO-1), which exerts cytoprotective effects in aging-related diseases, whereas the effect of CO on cellular senescence and aging has not been elucidated. In the current study, we clearly demonstrated that CO delays the process of cellular senescence and aging through regulation of miR-34a and Sirt1 expression. CO reduced H2O2-induced premature senescence in human ...
Publication date: Available online 1 April 2020Source: Journal of Microbiological MethodsAuthor(s): Wisnu Tafroji, Felicia Monica Bernadette, Ernawati A. Giri Rachman, Dodi Safari
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