Micro-RNA 18b and interleukin 17A profiles in relapsing remitting multiple sclerosis

ConclusionUpregulation of miR-18 during relapse in patients with RRMS points to a possible contribution to the pathogenesis of the inflammatory process in MS. The lack of a significant correlation between upregulated miR-18 and IL-17A implicates that the influence of miR-18 may be exhibited via another inflammatory pathway.
Source: Multiple Sclerosis and Related Disorders - Category: Neurology Source Type: research