Cholesterol: The Myth of Heart Disease
The next time your doctor says you need to lower your LDL cholesterol so you don’t have a heart attack or stroke, show him the results of the Northern Manhattan Study. The researchers that conducted the study found that higher LDL cholesterol was linked to LOWER stroke risk.1 And another study published this year reviewed research on nearly 70,000 people. The authors of that study found NO LINK between LDL cholesterol and premature deaths in people over 60 from heart disease.2 LDL Isn’t “Bad” Cholesterol This backs up what I’ve been saying for two decades! LDL isn’t “bad” cholesterol — and it’s not what causes heart disease or strokes. But despite the mounting evidence that’s turning conventional medicine’s cholesterol myth upside-down, don’t expect your doctor to change his tune anytime soon. If you have high cholesterol, your doctor will still likely try to put you on a statin drug. Especially if you have high LDL cholesterol. What are statin drugs? Statins are one of the most commonly prescribed drugs in the world. They’re a multibillion-dollar industry. Side Effects of Statins Nearly 30% of Americans over 40 are on one. And there’s been a push to up those numbers… even though statins cause dangerous side effects like liver damage and kidney failure. They can even kill you! Causes of Inflammation and Oxidation The truth is that heart disease and strokes are caused by...
This report continues the development of ALIs as a clinical tool in wildlife while systematically testing one possible method for determining an optimal ALI for a particular species. PMID: 32228119 [PubMed - as supplied by publisher]
Authors: Park J, Kim J, Chen Y, Song HC, Chen Y, Zheng M, Surh YJ, Kim UH, Park JW, Joe Y, Chung HT Abstract Oxidative stress is recognised as a key factor that can lead to cellular senescence and aging. Carbon monoxide (CO) is produced by haemoxygenase-1 (HO-1), which exerts cytoprotective effects in aging-related diseases, whereas the effect of CO on cellular senescence and aging has not been elucidated. In the current study, we clearly demonstrated that CO delays the process of cellular senescence and aging through regulation of miR-34a and Sirt1 expression. CO reduced H2O2-induced premature senescence in human ...
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