Low-grade endotoxemia in acute pulmonary embolism: Links with prothrombotic plasma fibrin clot phenotype

Lipopolysaccharide (LPS) can traverse the intestinal barrier and enter bloodstream, causing endotoxemia and triggering inflammation. Increased circulating LPS were reported in arterial thromboembolism. We investigated whether increased LPS occur in acute pulmonary embolism (PE) and if it is associated with a prothrombotic state.
Source: Thrombosis Research - Category: Hematology Authors: Tags: Full Length Article Source Type: research