METTL14 promotes the development of diabetic kidney disease by regulating m6A modification of TUG1

ConclusionMETTL14 promoted renal tubular epithelial cell apoptosis and ERS by activating MAPK/ERK pathway through m6A modification of TUG1, thereby accelerating DKD progression.
Source: Acta Diabetologica - Category: Endocrinology Source Type: research