Cigarette smoke extract-mediated FABP4 upregulation suppresses viability and induces apoptosis, inflammation and oxidative stress of bronchial epithelial cells by activating p38 MAPK/MK2 signaling pathway

Long-term inhalation of cigarette smoke is considered to be one of the main causes of bronchial epithelioid cell damage, but its underlying mechanism has to be further clarified.

Source: Journal of Inflammation - June 15, 2022 Category: Allergy & Immunology Authors: Wei Zhang, Yibin Zhang and Qi Zhu Tags: Research Source Type: research