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Total 2815 results found since Jan 2013.

The Effect of Corticosteroids on Sinus Microbiota in Chronic Rhinosinusitis Patients with Nasal Polyposis
CONCLUSION: In this pilot study, both treatment options were effective to improve symptoms in the short-term but not in the long-term, and were not linked to any clear sinus microbiota response. As a result, this study supports the avoidance of systemic antibiotics without evidence of active infection.PMID:37475202 | DOI:10.1177/19458924231183848
Source: American Journal of Rhinology and Allergy - July 21, 2023 Category: ENT & OMF Authors: Yousif Alammar Simon Rousseau Martin Desrosiers Marc A Tewfik Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research