H2S attenuates the myocardial fibrosis in diabetic rats through modulating PKC-ERK1/2MAPK signaling pathway.

CONCLUSION: The myocardial fibrosis in DM rats can be attenuated effectively by exogenous H2S, and the underlying mechanism is likely to regulating PKC-ERK1/2MAPK signaling pathway, improving the MMPs/TIMPs expression dysregulation and inhibiting inflammatory reaction. PMID: 31045549 [PubMed - as supplied by publisher]

https://www.ncbi.nlm.nih.gov/pubmed/31045549?dopt=Abstract

Source: Technology and Health Care - April 27, 2019 Category: Medical Devices Authors: Long J, Liu M, Liu S, Tang F, Tan W, Xiao T, Chu C, Yang J Tags: Technol Health Care Source Type: research