Is antacid use during pregnancy tied to childhood asthma?
(Reuters Health) - Pregnant women who take antacids may be more likely to have children who go on to develop asthma than their counterparts who don ’t take these medications, a research review suggests.
BOSTON (CBS) – Health officials say the flu season may be peaking which hopefully means that cases will soon start to decline. There’s some evidence that this is happening in Massachusetts but only time will tell. That said it is still not too late to get a flu shot so please get one. People with flu symptoms often wonder when they should just stay at home or when they should see a doctor. If you have underlying medical conditions like asthma or heart disease or if you’re pregnant and you think you have the flu, call your doctor. They may want to treat you with anti-viral medication. Otherwise healthy o...
CONCLUSIONS: Given the prevalence of prenatal APAP use and the importance of language development, these findings, if replicated, would suggest that pregnant women should limit their use of this analgesic during pregnancy. PMID: 29331486 [PubMed - as supplied by publisher]
Pregnant women who take antacids may be more likely to have children who go on to develop asthma than their counterparts who don ’ t take these medications, a research review suggests.Reuters Health Information
Taking acid-suppressive drugs during pregnancy is associated with elevated asthma risk among offspring, according to a meta-analysis in Pediatrics. Researchers assessed eight observational...
Conclusions Given the prevalence of prenatal APAP use and the importance of language development, these findings, if replicated, would suggest that pregnant women should limit their use of this analgesic during pregnancy.
Taking popular drugs for heartburn, a common complication of pregnancy, raised the risk of asthma in offspring.
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AbstractA bulk of literature demonstrated that respiratory allergy, and especially asthma, is prevalent in males during childhood, while it becomes more frequent in females from adolescence, i.e., after menarche, to adulthood. The mechanisms underlying the difference between females and males are the effects on the immune response of female hormones and in particular the modulation of inflammatory response by estrogens, as well as the result of the activity of various cells, such as dendritic cells, innate lymphoid cells, Th1, Th2, T regulatory (Treg) and B regulatory (Bregs) cells, and a number of proteins and cytokines, ...