Query: stroke

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Infectious Disease: Marburg Virus

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Total 6 results found since Jan 2013.

Health-related quality of life in patients with poststroke epilepsy
Conclusion Lesional epilepsy is associated with decreased HrQoL in patients with stroke. We identified HrQoL-determinants, which would improve the management of patients with poststroke epilepsy. These determinants include proper adjustment of AEDs with reduction of seizure frequency, treatment of depression, and focused rehabilitation programs for poststroke epilepsy.
Source: Epilepsy and Behavior - February 21, 2018 Category: Neurology Source Type: research

Health-related quality of life in patients with poststroke epilepsy.
CONCLUSION: Lesional epilepsy is associated with decreased HrQoL in patients with stroke. We identified HrQoL-determinants, which would improve the management of patients with poststroke epilepsy. These determinants include proper adjustment of AEDs with reduction of seizure frequency, treatment of depression, and focused rehabilitation programs for poststroke epilepsy. PMID: 29429905 [PubMed - as supplied by publisher]
Source: Epilepsy and Behaviour - February 8, 2018 Category: Neurology Authors: Winter Y, Daneshkhah N, Galland N, Kotulla I, Krüger A, Groppa S Tags: Epilepsy Behav Source Type: research

Factor VII activating protease (FSAP) regulates the expression of inflammatory genes in vascular smooth muscle and endothelial cells
The factor VII activating protease (FSAP) knockout mice have a bigger neointima after vascular injury and a larger infarct volume after stroke. The Marburg I (MI) single nucleotide polymorphism (SNP) in the FSAP-encoding gene is associated with an increased risk of stroke and carotid stenosis in humans. We hypothesize that the regulation of gene expression by FSAP in vascular cells accounts for its vasculo-regulatory properties.
Source: Atherosclerosis - August 25, 2017 Category: Cardiology Authors: Kristina Byskov, Thomas Boettger, Paul F. Ruehle, Nis Valentin Nielsen, Michael Etscheid, Sandip M. Kanse Source Type: research

Factor VII ‐activating protease deficiency promotes neointima formation by enhancing leukocyte accumulation
ConclusionsFSAP deficiency causes an increase in CCL2 expression and CCL2‐mediated infiltration of leukocytes into the injured vessel, thereby promoting SMC proliferation and migration by the activation of leukocyte‐derived gelatinases. These results provide a possible explanation for the observed association of the loss‐of‐function MI‐SNP with vascular proliferative diseases.
Source: Journal of Thrombosis and Haemostasis - September 7, 2016 Category: Hematology Authors: J. ‐M. Daniel, C. A. Reichel, T. Schmidt‐Woell, J. Dutzmann, G. Zuchtriegel, F. Krombach, J. Herold, J. Bauersachs, D. G. Sedding, S. M. Kanse Tags: Original Article Source Type: research

Factor VII activating protease deficiency promotes neointima formation by enhancing leukocyte accumulation.
CONCLUSIONS: FSAP deficiency causes an increase in CCL2 expression and CCL2-mediated infiltration of leukocytes into the injured vessel, thereby promoting SMC proliferation and migration by activation of leukocyte-derived gelatinases. These results provide a possible explanation for the observed association of the loss-of-function MI-SNP with vascular proliferative diseases. This article is protected by copyright. All rights reserved. PMID: 27431088 [PubMed - as supplied by publisher]
Source: Thrombosis and Haemostasis - July 19, 2016 Category: Hematology Authors: Daniel JM, Reichel CA, Schmidt-Woell T, Dutzmann J, Zuchtriegel G, Krombach F, Herold J, Bauersachs J, Sedding DG, Kanse SM Tags: J Thromb Haemost Source Type: research

Factor VII activating protease deficiency promotes neointima formation by enhancing leukocyte accumulation
ConclusionsFSAP deficiency causes an increase in CCL2 expression and CCL2‐mediated infiltration of leukocytes into the injured vessel, thereby promoting SMC proliferation and migration by activation of leukocyte‐derived gelatinases. These results provide a possible explanation for the observed association of the loss‐of‐function MI‐SNP with vascular proliferative diseases.This article is protected by copyright. All rights reserved.
Source: Journal of Thrombosis and Haemostasis - July 19, 2016 Category: Hematology Authors: Jan‐Marcus Daniel, Christoph A. Reichel, Thomas Schmidt‐Woell, Jochen Dutzmann, Gabriele Zuchtriegel, Fritz Krombach, Joerg Herold, Johann Bauersachs, Daniel G. Sedding, Sandip M. Kanse Tags: Original Article ‐ Vascular Biology Source Type: research