A Tongue Twister's Guide to Mastering Anatomy Pronunciation | Winter Shorts | TAPP 145
Episode 145 of The A&P Professor podcast is one of ourwinter shorts, where I replay interesting segments from previous episodes. In this one, you ' ll hear about the trials and tribulations of teaching and learningpronunciations of anatomy and physiology terminology. Including why the instructor is ALWAYS correct!00:00 | Introduction01:07 | Variations in Anatomy& Physiology Pronunciations10:24 | Say Anatomy& Physiology Terms Out Loud20:30 | Staying Connected★ If you cannot see or activate the audio player, go to:theAPprofessor.org/podcast-episode-145.html🏅 Apply for your credential (badge/certificate)...
Source: The A and P Professor - January 2, 2024 Category: Physiology Authors: Kevin Patton Source Type: blogs

Fight Aging! Newsletter, January 1st 2024
Discussion of What is Need to Speed the Pace at which Drugs to Treat Aging Arrive in the Clinic Cellular Senescence in the Aging Brain, a Contributing Cause of Cognitive Decline Reviewing What is Known of the Mechanisms of Taurine Supplementation Relevant to Aging and Metabolism Blunt Thoughts on Calculating the Revealed Value of Human Life A Look Back at 2023: Progress Towards the Treatment of Aging as a Medical Condition Towards Adjustment of the Gut Microbiome to Slow Aging Gene Therapy Enhances Object Recognition Memory in Young and Old Mice Benefits of Sem...
Source: Fight Aging! - December 31, 2023 Category: Research Authors: Reason Tags: Newsletters Source Type: blogs

This Personality Trait Is A Sign Of High Fluid Intelligence
Fluid intelligence refers to the raw speed at which the brain works. (Source: PsyBlog | Psychology Blog)
Source: PsyBlog | Psychology Blog - December 31, 2023 Category: Psychiatry & Psychology Authors: Jeremy Dean Tags: Intelligence Personality Source Type: blogs

A Look Back at 2023: Progress Towards the Treatment of Aging as a Medical Condition
The market has been in the doldrums and it has been a tough year for fundraising, both for non-profits and biotech startups. The conferences have exhibited more of an academic focus as companies tightened belts and postponed investment rounds, while investors stayed home. Not that this halts the flow of hype for some projects, and nor has it slowed media commentary on the longevity industry as it presently stands. A few of the articles in that commmentary are even interesting to read! The field has grown and is more mature now than has ever been the case. Biotech of all forms is a challenging field with a high failure rate...
Source: Fight Aging! - December 29, 2023 Category: Research Authors: Reason Tags: Of Interest Source Type: blogs

Econoclasm Chapter Two, continued: " Insurance "
 Health insurance – again I’m using the term because everybody else does, not because I think it’s accurate – can work in many ways. One of the most important broad dimensions is how the benefit gets delivered. ·Indemnity plans are the most like fire insurance. They pay money when the beneficiary incurs medical expenses. (The money could be paid to the beneficiary, or directly to the provider. That doesn ’t much matter.)·Service benefit plans have negotiated arrangements with providers to pay them a certain amount for a given service, when it is provided.·Service delivery plans actually provide the s...
Source: Stayin' Alive - December 28, 2023 Category: American Health Source Type: blogs

Amyloid- β Specific Regulatory T Cells to Treat Alzheimer's Disease
T cells of the adaptive immune system do find their way into the brain to some degree, even given the existence of the blood-brain barrier that separates the brain from the vasculature. Researchers here report on an effort to engineer regulatory T cells to recognize amyloid-β, associated with the onset of Alzheimer's disease. In an animal model of Alzheimer's disease, mice engineered to generate amyloid-β aggregates, these engineered regulatory T cells reduced the resulting pathology by migrating into the brain and dampening the maladaptive inflammatory responses characteristic of neurodegenerative conditions. R...
Source: Fight Aging! - December 28, 2023 Category: Research Authors: Reason Tags: Daily News Source Type: blogs

Reviewing What is Known of the Mechanisms of Taurine Supplementation Relevant to Aging and Metabolism
Taurine is a semi-essential amino acid. Dietary taurine supplementation has been shown to modestly slow aging in mice, though as for all such interventions there is always the question of whether it will prove to be less useful in humans, and also whether these results in mice will be disproved by the much more rigorous Interventions Testing Program (ITP), once that group gets around to assessing taurine supplementation. Few of the numerous interventions thought to modestly slow aging in mice on the basis of earlier research actually held up once subjected to the ITP degree of experimental rigor. Speculatively, taur...
Source: Fight Aging! - December 27, 2023 Category: Research Authors: Reason Tags: Medicine, Biotech, Research Source Type: blogs

Lifestyle Matters: Let ’s optimize cognition, health and life in 2024
Welcome to a new edition of SharpBrains e‑newsletter, featuring fascinating research findings on lifestyle, protective brain structures, Internet access, mental health, brain imaging, and more. #1. Lifestyle matters: What we can do in 2024 to optimize cognition and life, delaying cognitive problems even dementia “Actor Chris Hemsworth…watched his grandfather live with Alzheimer’s and is making lifestyle changes after learning he has two copies of the APOE4 gene. This gene is a risk factor for Alzheimer’s, and having two copies significantly increases his risk of developing the same condition.” (Smart move reg...
Source: SharpBrains - December 27, 2023 Category: Neuroscience Authors: SharpBrains Tags: Brain/ Mental Health SharpBrains Monthly eNewsletter Technology & Innovation adhd Alzheimer’s APOE4 gene Brain-Imaging dementia symptoms frontotemporal dementia lifestyle Neurophet Source Type: blogs

Ring In the New Year With Basic Research
Empowering basic biomedical research, which focuses on understanding how living systems work, is one of NIGMS’ main goals. This type of research not only helps us learn how our bodies and those of other organisms function but also lays the foundation for advances in disease diagnosis, treatment, and prevention. We’re excited to see what the upcoming year has in store for the field! In preparation, we’re highlighting what NIGMS-supported scientists had to say in 2023 about the many merits of basic research. Also check out the links to the Biomedical Beat posts that feature them if you haven’t already. ...
Source: Biomedical Beat Blog - National Institute of General Medical Sciences - December 27, 2023 Category: Research Authors: Chrissa Chverchko Tags: Being a Scientist Scientific Process Source Type: blogs

Toxic Tau Aggregates Alter Cell Nucleus Structure in Harmful Ways
The set of neurodegenerative diseases characterized by aggregation of altered tau protein are collectively known as tauopathies. Alzheimer's disease is the best known of these conditions. The later stage of Alzheimer's disease, in which cell death is widespread, is characterized by tau aggregation and chronic inflammation of brain tissue. As noted here, how exactly tau alteration and aggregation causes dysfunction is still an active area of research that may result in ways to sabotage the progression of tauopathies. Tauopathies are characterized by the buildup of tau inside the brain. Alzheimer's disease is well k...
Source: Fight Aging! - December 27, 2023 Category: Research Authors: Reason Tags: Daily News Source Type: blogs

2024 Prediction: Society Will Arrive at an Inflection Point in AI Advancement
By MIKE MAGEE For my parents, March, 1965 was a banner month. First, that was the month that NASA launched the Gemini program, unleashing “transformative capabilities and cutting-edge technologies that paved the way for not only Apollo, but the achievements of the space shuttle, building the International Space Station and setting the stage for human exploration of Mars.” It also was the last month that either of them took a puff of their favored cigarette brand – L&M’s. They are long gone, but the words “Gemini” and the L’s and the M’s have taken on new meaning and relevance now six decades l...
Source: The Health Care Blog - December 27, 2023 Category: Consumer Health News Authors: matthew holt Tags: Health Tech AI Google LLM Microsoft Mike Magee Source Type: blogs

Cellular Senescence in the Aging Brain, a Contributing Cause of Cognitive Decline
Senescent cells are created throughout the body at all stages of life, largely when somatic cells reach the Hayflick limit on replication. Senescent cells cease replication and begin to energetically produce pro-growth, pro-inflammatory factors, attracting the attention of the immune system and otherwise changing the behavior of surrounding cells. Cell stress and mutational damage can induce senescence, and in this case senescence is a mechanism that acts to limit the risk of cancer. Tissue injury also produces senescent cells, and here they help to coordinate the activities of the many different cell types that become inv...
Source: Fight Aging! - December 26, 2023 Category: Research Authors: Reason Tags: Medicine, Biotech, Research Source Type: blogs

10 Most Surprising Psychology Studies Of 2023
Can an old brain learn at the same rate as a young one? Do the selfish prefer to exploit the selfless? Some surprising answers to these, and more questions revealed. (Source: PsyBlog | Psychology Blog)
Source: PsyBlog | Psychology Blog - December 25, 2023 Category: Psychiatry & Psychology Authors: Jeremy Dean Tags: Most popular Source Type: blogs

Gene Therapy Enhances Object Recognition Memory in Young and Old Mice
In this study, we demonstrated that the same treatment with RGS14414 in visual area V2, which is relatively unaffected in neurodegenerative diseases such as Alzheimer's disease, produced long-lasting enhancement of ORM in young animals and prevent ORM deficits in rodent models of aging and Alzheimer's disease. Furthermore, we found that the prevention of memory deficits was mediated through the upregulation of neuronal arborization and spine density, as well as an increase in brain-derived neurotrophic factor (BDNF). A knockdown of BDNF gene in RGS14414-treated aging rats and Alzheimer's disease model mice caused complete ...
Source: Fight Aging! - December 25, 2023 Category: Research Authors: Reason Tags: Daily News Source Type: blogs