Corrigendum to Macrophage-derived inflammation promotes pulmonary vascular remodeling in hypoxia-induced pulmonary arterial hypertension mice [Immunology Letters 263 (2023) 113-122 /IMLET_6819]
Immunol Lett. 2024 Feb 23;266:106843. doi: 10.1016/j.imlet.2024.106843. Online ahead of print.NO ABSTRACTPMID:38401398 | DOI:10.1016/j.imlet.2024.106843 (Source: Immunology Letters)
Source: Immunology Letters - February 24, 2024 Category: Allergy & Immunology Authors: Hong Liu Source Type: research
Corrigendum to Macrophage-derived inflammation promotes pulmonary vascular remodeling in hypoxia-induced pulmonary arterial hypertension mice [Immunology Letters 263 (2023) 113-122 /IMLET_6819]
Immunol Lett. 2024 Feb 23;266:106843. doi: 10.1016/j.imlet.2024.106843. Online ahead of print.NO ABSTRACTPMID:38401398 | DOI:10.1016/j.imlet.2024.106843 (Source: Immunology Letters)
Source: Immunology Letters - February 24, 2024 Category: Allergy & Immunology Authors: Hong Liu Source Type: research
Corrigendum to Macrophage-derived inflammation promotes pulmonary vascular remodeling in hypoxia-induced pulmonary arterial hypertension mice [Immunology Letters 263 (2023) 113-122 /IMLET_6819]
Immunol Lett. 2024 Feb 23;266:106843. doi: 10.1016/j.imlet.2024.106843. Online ahead of print.NO ABSTRACTPMID:38401398 | DOI:10.1016/j.imlet.2024.106843 (Source: Immunology Letters)
Source: Immunology Letters - February 24, 2024 Category: Allergy & Immunology Authors: Hong Liu Source Type: research
Corrigendum to Macrophage-derived inflammation promotes pulmonary vascular remodeling in hypoxia-induced pulmonary arterial hypertension mice [Immunology Letters 263 (2023) 113-122 /IMLET_6819]
Immunol Lett. 2024 Feb 23;266:106843. doi: 10.1016/j.imlet.2024.106843. Online ahead of print.NO ABSTRACTPMID:38401398 | DOI:10.1016/j.imlet.2024.106843 (Source: Immunology Letters)
Source: Immunology Letters - February 24, 2024 Category: Allergy & Immunology Authors: Hong Liu Source Type: research
Corrigendum to Macrophage-derived inflammation promotes pulmonary vascular remodeling in hypoxia-induced pulmonary arterial hypertension mice [Immunology Letters 263 (2023) 113-122 /IMLET_6819]
Immunol Lett. 2024 Feb 23;266:106843. doi: 10.1016/j.imlet.2024.106843. Online ahead of print.NO ABSTRACTPMID:38401398 | DOI:10.1016/j.imlet.2024.106843 (Source: Immunology Letters)
Source: Immunology Letters - February 24, 2024 Category: Allergy & Immunology Authors: Hong Liu Source Type: research
Corrigendum to Macrophage-derived inflammation promotes pulmonary vascular remodeling in hypoxia-induced pulmonary arterial hypertension mice [Immunology Letters 263 (2023) 113-122 /IMLET_6819]
Immunol Lett. 2024 Feb 23;266:106843. doi: 10.1016/j.imlet.2024.106843. Online ahead of print.NO ABSTRACTPMID:38401398 | DOI:10.1016/j.imlet.2024.106843 (Source: Immunology Letters)
Source: Immunology Letters - February 24, 2024 Category: Allergy & Immunology Authors: Hong Liu Source Type: research
Corrigendum to Macrophage-derived inflammation promotes pulmonary vascular remodeling in hypoxia-induced pulmonary arterial hypertension mice [Immunology Letters 263 (2023) 113-122 /IMLET_6819]
Immunol Lett. 2024 Feb 23;266:106843. doi: 10.1016/j.imlet.2024.106843. Online ahead of print.NO ABSTRACTPMID:38401398 | DOI:10.1016/j.imlet.2024.106843 (Source: Immunology Letters)
Source: Immunology Letters - February 24, 2024 Category: Allergy & Immunology Authors: Hong Liu Source Type: research
Corrigendum to Macrophage-derived inflammation promotes pulmonary vascular remodeling in hypoxia-induced pulmonary arterial hypertension mice [Immunology Letters 263 (2023) 113-122 /IMLET_6819]
Immunol Lett. 2024 Feb 23;266:106843. doi: 10.1016/j.imlet.2024.106843. Online ahead of print.NO ABSTRACTPMID:38401398 | DOI:10.1016/j.imlet.2024.106843 (Source: Immunology Letters)
Source: Immunology Letters - February 24, 2024 Category: Allergy & Immunology Authors: Hong Liu Source Type: research
Aged A βPPswe/PS1ΔE9 mice as a useful animal model for studying the link between immunological senescence and diseases
This study aimed to elucidate the changes in systemic characteristics aside from those associated with AD between elderly APPswe/PS1ΔE9 mice and littermate control wild-type mice. Tumors in all organs were considerably more frequent in AD mice aged 24 months than in the control wild-type mice. In addition, the survival rate of aged AD mice was considerably lower than that of wild-type control mice. Further, we discovered that the phenotypic difference was mainly caused by severe immunological aging, as evidenced by a high proportion of exhausted T lymphocytes in AD mice compared to wild-type mice of the same age. Based on...
Source: Immunology Letters - February 14, 2024 Category: Allergy & Immunology Authors: Jing Luan Na Guo Fengrui Hu Xingchun Gou Lixian Xu Source Type: research
Aged A βPPswe/PS1ΔE9 mice as a useful animal model for studying the link between immunological senescence and diseases
This study aimed to elucidate the changes in systemic characteristics aside from those associated with AD between elderly APPswe/PS1ΔE9 mice and littermate control wild-type mice. Tumors in all organs were considerably more frequent in AD mice aged 24 months than in the control wild-type mice. In addition, the survival rate of aged AD mice was considerably lower than that of wild-type control mice. Further, we discovered that the phenotypic difference was mainly caused by severe immunological aging, as evidenced by a high proportion of exhausted T lymphocytes in AD mice compared to wild-type mice of the same age. Based on...
Source: Immunology Letters - February 14, 2024 Category: Allergy & Immunology Authors: Jing Luan Na Guo Fengrui Hu Xingchun Gou Lixian Xu Source Type: research
Aged A βPPswe/PS1ΔE9 mice as a useful animal model for studying the link between immunological senescence and diseases
This study aimed to elucidate the changes in systemic characteristics aside from those associated with AD between elderly APPswe/PS1ΔE9 mice and littermate control wild-type mice. Tumors in all organs were considerably more frequent in AD mice aged 24 months than in the control wild-type mice. In addition, the survival rate of aged AD mice was considerably lower than that of wild-type control mice. Further, we discovered that the phenotypic difference was mainly caused by severe immunological aging, as evidenced by a high proportion of exhausted T lymphocytes in AD mice compared to wild-type mice of the same age. Based on...
Source: Immunology Letters - February 14, 2024 Category: Allergy & Immunology Authors: Jing Luan Na Guo Fengrui Hu Xingchun Gou Lixian Xu Source Type: research
Aged A βPPswe/PS1ΔE9 mice as a useful animal model for studying the link between immunological senescence and diseases
This study aimed to elucidate the changes in systemic characteristics aside from those associated with AD between elderly APPswe/PS1ΔE9 mice and littermate control wild-type mice. Tumors in all organs were considerably more frequent in AD mice aged 24 months than in the control wild-type mice. In addition, the survival rate of aged AD mice was considerably lower than that of wild-type control mice. Further, we discovered that the phenotypic difference was mainly caused by severe immunological aging, as evidenced by a high proportion of exhausted T lymphocytes in AD mice compared to wild-type mice of the same age. Based on...
Source: Immunology Letters - February 14, 2024 Category: Allergy & Immunology Authors: Jing Luan Na Guo Fengrui Hu Xingchun Gou Lixian Xu Source Type: research
Aged A βPPswe/PS1ΔE9 mice as a useful animal model for studying the link between immunological senescence and diseases
This study aimed to elucidate the changes in systemic characteristics aside from those associated with AD between elderly APPswe/PS1ΔE9 mice and littermate control wild-type mice. Tumors in all organs were considerably more frequent in AD mice aged 24 months than in the control wild-type mice. In addition, the survival rate of aged AD mice was considerably lower than that of wild-type control mice. Further, we discovered that the phenotypic difference was mainly caused by severe immunological aging, as evidenced by a high proportion of exhausted T lymphocytes in AD mice compared to wild-type mice of the same age. Based on...
Source: Immunology Letters - February 14, 2024 Category: Allergy & Immunology Authors: Jing Luan Na Guo Fengrui Hu Xingchun Gou Lixian Xu Source Type: research
Aged A βPPswe/PS1ΔE9 mice as a useful animal model for studying the link between immunological senescence and diseases
This study aimed to elucidate the changes in systemic characteristics aside from those associated with AD between elderly APPswe/PS1ΔE9 mice and littermate control wild-type mice. Tumors in all organs were considerably more frequent in AD mice aged 24 months than in the control wild-type mice. In addition, the survival rate of aged AD mice was considerably lower than that of wild-type control mice. Further, we discovered that the phenotypic difference was mainly caused by severe immunological aging, as evidenced by a high proportion of exhausted T lymphocytes in AD mice compared to wild-type mice of the same age. Based on...
Source: Immunology Letters - February 14, 2024 Category: Allergy & Immunology Authors: Jing Luan Na Guo Fengrui Hu Xingchun Gou Lixian Xu Source Type: research
Aged A βPPswe/PS1ΔE9 mice as a useful animal model for studying the link between immunological senescence and diseases
This study aimed to elucidate the changes in systemic characteristics aside from those associated with AD between elderly APPswe/PS1ΔE9 mice and littermate control wild-type mice. Tumors in all organs were considerably more frequent in AD mice aged 24 months than in the control wild-type mice. In addition, the survival rate of aged AD mice was considerably lower than that of wild-type control mice. Further, we discovered that the phenotypic difference was mainly caused by severe immunological aging, as evidenced by a high proportion of exhausted T lymphocytes in AD mice compared to wild-type mice of the same age. Based on...
Source: Immunology Letters - February 14, 2024 Category: Allergy & Immunology Authors: Jing Luan Na Guo Fengrui Hu Xingchun Gou Lixian Xu Source Type: research
