American Journal of Rhinology and Allergy This is an RSS file. You can use it to subscribe to this data in your favourite RSS reader or to display this data on your own website or blog.

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research

Crocin Inhibits the Type 2 Inflammatory Response Produced by ILC2s in Eosinophilic Nasal Polyps
CONCLUSION: Crocin inhibited type 2 inflammation induced by ILC2 activation at low concentrations via inhibiting the activation of NF-κB.PMID:37424236 | DOI:10.1177/19458924231185296 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Xu Xiaodong Li Tao Liu Jianmin Zhou Jing Zhong Bing Du Jintao Claus Bachert Ba Luo Source Type: research

Cigarette Smoke Mediates Nasal Epithelial Barrier Dysfunction via TNF- α
CONCLUSIONS: We found that cigarette smoke disrupted the nasal mucosal barrier, and the extent of the damage was correlated with the duration of cigarette smoke exposure. We showed that TNF-α can disrupt the continuity and attenuate the expression of tight junction proteins in human bronchial epithelial cells. Therefore, cigarette smoke may induce nasal epithelial barrier dysfunction through TNF-α.PMID:37424240 | DOI:10.1177/19458924231184741 (Source: American Journal of Rhinology and Allergy)
Source: American Journal of Rhinology and Allergy - July 10, 2023 Category: ENT & OMF Authors: Ju Guo Xuan Meng Yao-Ming Zheng Shan-Kun Zhao Chen Qiang Li-Bo Zhou Source Type: research