Promotion of miR-221-5p on the Sensitivity of Gastric Cancer Cells to Cisplatin and Its Effects on Cell Proliferation and Apoptosis by Regulating DDR1 Retraction
STOMACH CANCER has many different digestive symptoms, but can easily be mistaken for other conditions - here's when you should see a doctor.
SCIENTIFIC advancements in cancer research have made significant gains, helping millions receive curative treatment. But academics continue to stress that prevention is better than cure. One pill which is widely prescribed in the UK could increase the risk of stomach cancer twofold.
In conclusion, circ_0003159 inhibited GC development in vitro and in vivo by enriching the level of LIFR via direct binding to miR-221-3p/miR-222-3p.
ConclusionsPatient experiences related toH. pylori management are predominantly negative. Increasing providers ’ awareness about patients’ values, beliefs, anxieties, and expectations surroundingH. pylori diagnosis/treatment may improve provider-patient communication and, ideally, related outcomes.
CANCEROUS cells multiplying in the stomach can lead to notoriously "hard to spot" symptoms. However, once you are aware of the warning signs, you can seek medical assistance should they arise.
CONCLUSIONS: The CT-based AI model demonstrated good performance for diagnosing SRCC, stratifying patient prognosis, and predicting chemotherapy responses. Advanced SRCC patients with AI-predicted low-risk status may benefit substantially from adjuvant chemotherapy. This article is protected by copyright. All rights reserved.PMID:35032039 | DOI:10.1002/mp.15437
CONCLUSIONS: The ERAS protocol could be safely used in elderly gastric cancer patients undergoing gastrectomy and shorten postoperative hospital stay.PMID:35031180 | DOI:10.1016/j.asjsur.2021.10.040
CONCLUSION: Kinase amplifications could be used as diagnostic, prognostic, and predictive biomarkers in various cancer types. Hence targeting kinase and related signalling molecules could be considered as a potential approach to prevent cancer through these organisms. Here we summarize the brief information about the role of kinases, signalling and their therapeutics in GC concerning H. pylori, EBV and HCMV.PMID:35031925 | DOI:10.1007/s11033-021-07063-9
ConclusionsAIG induced aberrant DNA methylation in gastric mucosa. However, the degree of DNA methylation was less than that by HPG, which reflected carcinogenic risk.