Emerging perspectives on mitochondrial dysfunctioning and inflammation in epileptogenesis

ConclusionThe current review highlights the further opportunity for establishing therapeutic interventions underlying the apparent correlation of neuroinflammation mediated mitochondrial oxidative stress might contribute to common intracellular mechanisms underlying a future prospective of drug treatment targeting mitochondrial dysfunction linked to the neuroinflammation in epilepsy.
Source: Inflammation Research - Category: Research Source Type: research