Active components from Lagotis brachystachya maintain uric acid homeostasis by inhibiting renal TLR4-NLRP3 signaling in hyperuricemic mice

In conclusion, these results indicated that luteolin, luteoloside and apigenin could attenuate hyperuricemia by decreasing the production and increasing the excretion of uric acid, which were mediated by inhibiting inflammatory signaling pathways.

http://link.springer.com/10.1007/s10787-021-00844-5

Source: Inflammopharmacology - July 9, 2021 Category: Drugs & Pharmacology Source Type: research