Thromboembolic involvement and its possible pathogenesis in COVID-19 mortality: lesson from post-mortem reports

In conclusion, a complex interaction of SARS-CoV-2 virus invasion with platelets, leukocytes, endothelial cells, inflammation, immune response, and the possible involvement of megakaryocytes may increase the cumulative risk of thrombosis by a yet unclear cellular and humoral interaction.PMID:33629337 | DOI:10.26355/eurrev_202102_24878

Source: European Review for Medical and Pharmacological Sciences - February 25, 2021 Category: Drugs & Pharmacology Authors: K Dwiputra Hernugrahanto D Novembri Utomo H Hariman N C Budhiparama D Medika Hertanto D Santoso P C W Hogendoorn Source Type: research