miR-558 Reduces the Damage of HBE Cells Exposed to Cigarette Smoke Extract by Targeting TNFRSF1A and Inactivating TAK1/MAPK/NF- κB Pathway.

CONCLUSIONS: Up-regulation of miR-558 relieved the damage of HBE cells-triggered by CSE via reducing TNFRSF1A and inactivating TAK1/MAPK/NF-κB pathway, affording novel molecules for COPD treatment. PMID: 33459100 [PubMed - as supplied by publisher]

https://www.ncbi.nlm.nih.gov/pubmed/33459100?dopt=Abstract

Source: Immunological Investigations - January 21, 2021 Category: Allergy & Immunology Tags: Immunol Invest Source Type: research

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