Modulation of pancreatic neuroendocrine neoplastic cell fate by autophagy mediated death

Conclusion Our study h ighlights that the autophagy functional restoration and prolongation of this catabolic process, mediated by inhibition of deacetylase, is responsible for the reduction of pNEN cells. Prompting of autophagy cell death could be a promising strategy for the therapy of pNEN.
Source: Neuroendocrinology - Category: Endocrinology Source Type: research