Metabolic reprogramming as a key regulator in the pathogenesis of rheumatoid arthritis

This article reviews the metabolic reprogramming of immune cells and fibroblast-like synoviocytes in RA and their relationship to mitochondrial disfunction, as well as the key pro-inflammatory pathways associated with metabolic reprogramming and chemotherapy as a potential future therapeutic strategy for RA.
Source: Inflammation Research - Category: Research Source Type: research