Distinguishing Behavioral Variant FTD From Psychiatric Disorder Distinguishing Behavioral Variant FTD From Psychiatric Disorder

Diagnosis of the behavioral variant of frontotemporal dementia remains challenging. This review presents clinical recommendations for differentiating bvFTD from primary psychiatric disorders.Brain
Source: Medscape Radiology Headlines - Category: Radiology Tags: Neurology & Neurosurgery Journal Article Source Type: news

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Charities are using tailored music to bring back memories for dementia patients from ethnic minorities.
Source: BBC News | Health | UK Edition - Category: Consumer Health News Source Type: news
ConclusionsModules of tightly regulated lipids and proteins, drivers in lipid homeostasis and innate immunity, are strongly associated with AD phenotypes.
Source: Translational Neurodegeneration - Category: Neurology Source Type: research
Dear Carol: My dad, 72, has been adventurous all his life and that spirit doesn’t seem to be dampened by aging or even some health problems. He’s always loved snow skiing though thankfully he settles for just cross-country skiing on our flat trails. He still wants to ride his bike in all seasons except winter, and that scares me to death.  He says this is healthy. Yes, but what if he falls doing these things? How do I convince him that he’s risking life and limb with the way he lives? His brain seems to be working fine so I’m limited in how much I can intervene, but I live in fear that he will ...
Source: Minding Our Elders - Category: Geriatrics Authors: Source Type: blogs
Publication date: Available online 20 September 2020Source: Journal of Biomedical InformaticsAuthor(s): Fadi Thabtah, David Peebles, Jenny Retzler, Chanchala Hathurusingha
Source: Journal of Biomedical Informatics - Category: Information Technology Source Type: research
Fight Aging! publishes news and commentary relevant to the goal of ending all age-related disease, to be achieved by bringing the mechanisms of aging under the control of modern medicine. This weekly newsletter is sent to thousands of interested subscribers. To subscribe or unsubscribe from the newsletter, please visit: https://www.fightaging.org/newsletter/ Longevity Industry Consulting Services Reason, the founder of Fight Aging! and Repair Biotechnologies, offers strategic consulting services to investors, entrepreneurs, and others interested in the longevity industry and its complexities. To find out m...
Source: Fight Aging! - Category: Research Authors: Tags: Newsletters Source Type: blogs
Conditions:   Dementia;   Dementia, Vascular;   Dementia, Mixed;   Dementia With Lewy Bodies;   Dementia Frontal;   Dementia Severe;   Dementia Moderate;   Dementia of Alzheimer Type Intervention:   Other: Advanced Cognitive Stimulation Therapy Hong Kong Sponsors:   University College, London;   The University of Hong Kong Not yet recruiting
Source: ClinicalTrials.gov - Category: Research Source Type: clinical trials
Condition:   Alzheimer Disease Interventions:   Behavioral: Standard Environmental Design;   Behavioral: Individual Environmental Design;   Behavioral: Placebo Sponsor:   Elizabeth K Rhodus Not yet recruiting
Source: ClinicalTrials.gov - Category: Research Source Type: clinical trials
Conditions:   Dementia;   Alzheimer Disease Intervention:   Behavioral: Care Ecosystem Sponsor:   Mclean Hospital Not yet recruiting
Source: ClinicalTrials.gov - Category: Research Source Type: clinical trials
Publication date: Available online 19 September 2020Source: Molecular and Cellular NeuroscienceAuthor(s): Georgie Lines, Jackie M. Casey, Elisavet Preza, Selina Wray
Source: Molecular and Cellular Neuroscience - Category: Neuroscience Source Type: research
We appreciate the criticism raised by Dr. Matsuura questioning the genetic evaluation for NOP56 gene of Asidan (spinocerebellar ataxia 36) showing clinical anticipation. Asidan is an autosomal dominant neurodegenerative disorder caused by a hexanucleotide GGCCTG repeat expansion in intron 1 of the NOP56 gene, showing cerebellar ataxia, motor neuron disease phenotype, hearing loss and frontal cognitive impairment [1 –3]. The NOP56 genetic mutation in Asidan is similar to the hexanucleotide GGGGCC repeat expansion in intron 1 of the C9orf72 gene observed in familial amyotrophic lateral sclerosis/frontotemporal dementia...
Source: Journal of the Neurological Sciences - Category: Neurology Authors: Tags: Letter to the Editor Source Type: research
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