iNOS is not responsible for RyR1 S-nitrosylation in mdx mice with truncated dystrophin

Previous research indicated that nitric oxide synthase (NOS) is the key molecule for S-nitrosylation of ryanodine receptor 1 (RyR1) in DMD model mice (mdx mice) and that both neuronal NOS (nNOS) and inducible NOS...

Source: BMC Musculoskeletal Disorders - July 21, 2020 Category: Orthopaedics Authors: Ken ’ichiro Nogami, Yusuke Maruyama, Ahmed Elhussieny, Fusako Sakai-Takemura, Jun Tanihata, Jun-ichi Kira, Yuko Miyagoe-Suzuki and Shin’ichi Takeda Tags: Research article Source Type: research