Cross talk between alcohol-induced oxidative stress and HCV replication.

In conclusion, acute alcohol treatment might contribute to increase HCV replication by interference in innate immunity and induction of autophagy. Chronic alcohol treatment caused oxidative stress, which disrupts autophagy and thereby increased the rate of Huh7 cell injury. PMID: 32448963 [PubMed - as supplied by publisher]

https://www.ncbi.nlm.nih.gov/pubmed/32448963?dopt=Abstract

Source: Archives of Microbiology - May 23, 2020 Category: Microbiology Authors: Sobhanimonfared F, Bamdad T, Roohvand F Tags: Arch Microbiol Source Type: research