AGR2-induced glucose metabolism facilitated the progression of endometrial carcinoma via enhancing the MUC1/HIF-1 α pathway

In conclusion, AGR2 was overexpressed in endometrial canc er and AGR2-induced glucose metabolism facilitated the progression of endometrial carcinoma via the MUC1/HIF-1α pathway. AGR2 may be an effective therapeutic target for endometrial carcinoma.

http://link.springer.com/10.1007/s13577-020-00356-4

Source: Human Cell - April 17, 2020 Category: Cytology Source Type: research