Effects of respiratory rate on venous-to-arterial CO 2 tension difference in septic shock patients undergoing volume mechanical ventilation

ConclusionsIn septic patients undergoing ventilation, respiratory alkalosis induced by hyperventilation caused an increase in the gapCO2. Clinicians should cautiously interpret the gapCO2 in hemodynamically stable ventilated septic shock patients and its relationship with low cardiac output and inadequate perfusion.
Source: European Journal of Medical Research - Category: Research Source Type: research