RapaLink-1 plays an antithrombotic role in antiphospholipid syndrome by improving autophagy both in vivo and vitro.

CONCLUSION: Our results revealed that Rapalink-1 has a potential to inhibit the formation of thrombus plaque in APS and these effects were dependent on facilitating cell autophagy both in vivo and in vitro. PMID: 32093890 [PubMed - as supplied by publisher]

https://www.ncbi.nlm.nih.gov/pubmed/32093890?dopt=Abstract

Source: Biochemical and Biophysical Research communications - February 21, 2020 Category: Biochemistry Authors: Mu F, Jiang Y, Ao F, Wu H, You Q, Chen Z Tags: Biochem Biophys Res Commun Source Type: research