Endocrine disruptors and thyroid autoimmunity
Publication date: Available online 11 February 2020Source: Best Practice &Research Clinical Endocrinology &MetabolismAuthor(s): Salvatore Benvenga, Giusy Elia, Francesca Ragusa, Sabrina Rosaria Paparo, Mattia Maria Sturniolo, Silvia Martina Ferrari, Alessandro Antonelli, Poupak FallahiAbstractMany papers evaluated the effect of the environmental, or occupational endocrine disruptors (ED), on the thyroid gland, that can lead to thyroid autoimmunity. A higher prevalence of “autoimmune thyroid diseases” (AITD) was observed in people living in polluted areas near to petrochemical plants, and in petrochemical workers, but also in area contaminated with organochlorine pesticides, or with polychlorinated biphenyls, or near aluminum foundries. The exposure to Hg in chloralkali workers, or in swordfish consumers has been also found to increase AITD prevalence. Vanadium has been shown to increase the inflammatory response of thyrocytes. A beneficial effect of omega-3 fatty acids, and of myo-inositol and selenomethionine have been shown to counteract the appearance of AITD in subjects exposed to environmental or occupational ED.More large studies are needed to investigate the potential roles of ED in the induction of AITD, and of agents or habits that are able to prevent them.
This report continues the development of ALIs as a clinical tool in wildlife while systematically testing one possible method for determining an optimal ALI for a particular species. PMID: 32228119 [PubMed - as supplied by publisher]
Authors: Park J, Kim J, Chen Y, Song HC, Chen Y, Zheng M, Surh YJ, Kim UH, Park JW, Joe Y, Chung HT Abstract Oxidative stress is recognised as a key factor that can lead to cellular senescence and aging. Carbon monoxide (CO) is produced by haemoxygenase-1 (HO-1), which exerts cytoprotective effects in aging-related diseases, whereas the effect of CO on cellular senescence and aging has not been elucidated. In the current study, we clearly demonstrated that CO delays the process of cellular senescence and aging through regulation of miR-34a and Sirt1 expression. CO reduced H2O2-induced premature senescence in human ...
Publication date: Available online 1 April 2020Source: Journal of Microbiological MethodsAuthor(s): Wisnu Tafroji, Felicia Monica Bernadette, Ernawati A. Giri Rachman, Dodi Safari
Publication date: Available online 2 April 2020Source: Journal of Microbiology, Immunology and InfectionAuthor(s): Chun-Min Kang, Xiang-Jun Chen, Ching-Chin Chih, Chen-Ching Hsu, Ping-Hung Chen, Tai fen Lee, Lee-Jene Teng, Po-Ren Hsueh
Publication date: Available online 1 April 2020Source: Journal of Microbiology, Immunology and InfectionAuthor(s): Chien-Yu Lee, Pi-Sheng Wang, Yuan-Der Huang, Yung-Ching Lin, Yung-Nien Hsu, Shih-Chung Chen
Publication date: Available online 1 April 2020Source: Journal of Microbiology, Immunology and InfectionAuthor(s): Chia-Hung Liao, Shih-Chang Hung, Yuan-Ti Lee, Hung-Chang Hung, Po-Ren Hsueh
Publication date: Available online 2 April 2020Source: Statistics &Probability LettersAuthor(s): Junhao Guo, Jie Zhou, Sanfeng Hu
Publication date: Available online 1 April 2020Source: Statistics &Probability LettersAuthor(s): S. Mousavinasr, C.R. Gonçalves, C.C.Y. Dorea
Publication date: 1 May 2020Source: Sensors and Actuators A: Physical, Volume 306Author(s): Chuang Ge, Leixiang Bian, Jiayang Li, Mingyou Zhong, Songtong Han, Yunfei Jia
Publication date: Available online 1 April 2020Source: Results in PhysicsAuthor(s): U.S. Okorie, A.N. Ikot, E.O. Chukwuocha, G.J. Rampho