GV1001 inhibits cell viability and induces apoptosis in castration-resistant prostate cancer cells through the AKT/NF- κ B/VEGF pathway

Conclusions: Our data demonstrated that GV1001 inhibited cell viability, induced apoptosis, and inhibited angiogenesis in CRPC cells by inhibition of the AKT/NF-κB/VEGF signaling pathway.
Source: Journal of Cancer - Category: Cancer & Oncology Authors: Tags: Research Paper Source Type: research