S100A8/A9 as a therapeutic target in myocardial infarction: cellular mechanisms, molecular interactions, and translational challenges

This editorial refers to ‘Inhibition of pro-inflammatory myeloid cell responses by short-term S100A9 blockade improves cardiac function after myocardial infarction’††, by G. Marinkovi ćet al., on page2713.
Source: European Heart Journal - Category: Cardiology Source Type: research