Microbiota-driven gut vascular barrier disruption is a prerequisite for non-alcoholic steatohepatitis development
Fatty liver disease is characterized by a series of pathological conditions ranging from hepatic lipid accumulation (steatosis), to hepatocyte degeneration (ballooning), inflammation (steatohepatitis) and, eventually, cirrhosis and hepatocellular carcinoma [1,2]. Fatty liver disease may be the result of long-term excessive ethanol consumption, (alcoholic liver disease), or of visceral obesity and metabolic syndrome without ethanol consumption, leading to nonalcoholic fatty liver disease (NAFLD) which can evolve to nonalcoholic steatohepatitis (NASH) [3].
Source: Journal of Hepatology - Category: Gastroenterology Authors: Juliette Mouries, Paola Brescia, Alessandra Silvestri, Ilaria Spadoni, Marcel Sorribas, Reiner Wiest, Erika Mileti, Marianna Galbiati, Pietro Invernizzi, Luciano Adorini, Giuseppe Penna, Maria Rescigno Source Type: research
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