Endogenous AT1 receptor-protein kinase C activity in the hypothalamus augments glutamatergic input and sympathetic outflow in hypertension.
This article is protected by copyright. All rights reserved.
PMID: 31241170 [PubMed - as supplied by publisher]
Source: The Journal of Physiology - Category: Physiology Authors: Ma H, Chen SR, Chen H, Pan HL Tags: J Physiol Source Type: research
More News: Brain | Cozaar | Genetics | Hyperactivity | Hypertension | Losartan | Neurology | Physiology | Reflex Sympathetic Dystrophy