Update: Have BIO2010 Goals Been Achieved?
Leaders in biology education are celebrating the progress made since the publication of BIO2010: Transforming Undergraduate Education for Future Research Biologists. The landmark report, published in 2003 by the National Research Council (NRC), called on institutions of higher education to revamp both the curricula and teaching methods in the life sciences to meet the challenges of the 21st century. BIO2010 urged much deeper connections between the biological sciences and mathematics, the physical sciences, and computer science. In addition, the report called on faculty to move out of the lecture hall and into the field and lab to help develop hands-on learning and higher-order thinking in their students. "There's been an incredible response," says John Jungck, vice president of the International Union of Biological Sciences and professor of biology at Beloit College. "One of the real highlights has been the collaboration between mathematicians and biologists in developing courses, majors, labs, and undergraduate research programs." Claudia Neuhauser, director of graduate studies in biomedical informatics and computational biology and vice chancellor of the University of Minnesota, Rochester, credits BIO2010 with influencing curricula around the country. "It's viewed as a very critical piece in catalyzing change in biology education," she says. Instead of using lectures to cram all of biology into students' first year, she says, professors more f...
This report continues the development of ALIs as a clinical tool in wildlife while systematically testing one possible method for determining an optimal ALI for a particular species. PMID: 32228119 [PubMed - as supplied by publisher]
Authors: Park J, Kim J, Chen Y, Song HC, Chen Y, Zheng M, Surh YJ, Kim UH, Park JW, Joe Y, Chung HT Abstract Oxidative stress is recognised as a key factor that can lead to cellular senescence and aging. Carbon monoxide (CO) is produced by haemoxygenase-1 (HO-1), which exerts cytoprotective effects in aging-related diseases, whereas the effect of CO on cellular senescence and aging has not been elucidated. In the current study, we clearly demonstrated that CO delays the process of cellular senescence and aging through regulation of miR-34a and Sirt1 expression. CO reduced H2O2-induced premature senescence in human ...
Publication date: Available online 1 April 2020Source: Journal of Microbiological MethodsAuthor(s): Wisnu Tafroji, Felicia Monica Bernadette, Ernawati A. Giri Rachman, Dodi Safari
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