Female anatomy and hysterical duality.
This article attempts to add another layer to our understanding of the phenomenon of hysterical duality. The author postulates that hysterical duality can be explained based on the dual-aspect model of feminine sexuality, which exhibits two initially contradictory paths: one derived from primary vaginal sensations and the other from clitoral pleasure. At first, these two paths create a fundamental split between representations of internal space, containment and motherhood and representations related to auto-eroticism and the effacement of the Other's presence and needs. The author argues that this manifest contradiction makes the attainment of integration in feminine development an intricate and protracted process, which involves an act of inversion. This inversion entails a post-Oedipal disavowal of primary vaginal sexuality, pending its rediscovery through the encounter with the Other. Hysteria is thus viewed as the result of a failure to perform this inversion and an inability to extract oneself from the position of a "Vaginal Girl", who defines herself through the desire of the other. This pathological course of development leaves the hysteric's sexuality in a split state and traps her in the duality of clitoral pleasure versus penetration, which unconsciously represents humiliation and exploitation. PMID: 30733551 [PubMed - as supplied by publisher]
Publication date: Available online 14 July 2020Source: British Journal of Oral and Maxillofacial SurgeryAuthor(s): L. Khojastepour, M. Moannaei, H.R. Eftekharian, M.S. Khaghaninejad, M. Mahjoori-Ghasrodashti, S. Tavanafar
Publication date: Available online 15 July 2020Source: Diabetes &Metabolic Syndrome: Clinical Research &ReviewsAuthor(s): M.G. Rajanandh
Publication date: Available online 13 July 2020Source: Canadian Journal of DiabetesAuthor(s): Yiting Wang, Fang Liu
Conclusion: This is the first study that reported the altered expression of exosomal miR-34a among children and adolescents with T1DM. Moreover, association of miR-34a with markers of dyslipidemia and endothelial dysfunction was identified, suggesting that it could play a role in regulation of lipid metabolism and endothelial function in T1DM. PMID: 32654473 [PubMed - as supplied by publisher]
Authors: Dourado M, Cavalcanti F, Vilar L, Cantilino A Abstract CKD has a high prevalence worldwide, mainly due to its main etiologies-diabetes and hypertension. It has high cardiovascular morbidity and mortality, with traditional risk factors such as atherosclerosis, hypertension, diabetes, smoking, and left ventricular hypertrophy being common. Nontraditional cardiovascular risk factors, such as anemia, hyperparathyroidism, chronic inflammation, and microalbuminuria, are also well studied. Prolactin is a hormone not only related to lactation but also being considered a uremic toxin by some authors. It accumulates...
Erratum to "Gastroenteropancreatic Neuroendocrine Neoplasia Characterization in Portugal: Results from the NETs Study Group of the Portuguese Society of Endocrinology, Diabetes and Metabolism". Int J Endocrinol. 2020;2020:9184324 Authors: Santos AP, Vinagre J, Soares P, Claro I, Sanches AC, Gomes L, Fernandes I, Catarino AL, Preto J, Pereira BD, Marques AP, Rodrigues F, Amaral C, Rocha G, Mellidez JC, Simões H, Lopes JM, Bugalho MJ, On Behalf Of The NETs Study Group Of The Portuguese Society Of Endocrinology Diabetes And Metabolism Abstract [This corrects the article DOI: 10.1155/2019/...
Conclusions: Maternal serum TG/HDL levels at late gestation are positively associated with neonatal body weight and the risk of macrosomia in women without DM. Maintaining maternal lipid levels in an appropriate range is important in the context of fetal overgrowth and primary prevention of macrosomia. PMID: 32655633 [PubMed]
Authors: Sun Z, Li P, Wang X, Lai S, Qiu H, Chen Z, Hu S, Yao J, Shen J Abstract As the major cause of female anovulatory infertility, polycystic ovary syndrome (PCOS) affects a great proportion of women at childbearing age. Although glucagon-like peptide 1 receptor agonists (GLP-IRAs) show therapeutic effects for PCOS, its target and underlying mechanism remains elusive. In the present study, we identified that, both in vivo and in vitro, GLP-1 functioned as the regulator of proliferation and antiapoptosis of MGCs of follicle in PCOS mouse ovary. Furthermore, forkhead box protein O1 (FoxO1) plays an important role...
Publication date: Available online 14 July 2020Source: Seminars in Cancer BiologyAuthor(s): Rui Caetano Oliveira, Henrique Alexandrino, Maria Augusta Cipriano, Filipe Caseiro Alves, José Guilherme Tralhão
Publication date: Available online 13 July 2020Source: Journal of Bone OncologyAuthor(s): Xiaomeng Li, Ning Wu, Wenjie Zhang, Ying Liu, Yue Ming