ESTROGEN RECEPTOR α and ARYL HYDROCARBON RECEPTOR CROSS-TALK in a TRANSFECTED HEPATOMA CELL LINE (HEPG2) EXPOSED TO 2,3,7,8-TETRACHLORODIBENZO-P-DIOXIN

This study provides new evidence of a TCDD-activated AhR-mediated molecular AhR/ERα cross-talk mechanism at transcriptional level via indirect inhibition of ERα and enhanced transcriptional activity of AhR in HepG2 cells.
Source: Toxicology Reports - Category: Toxicology Source Type: research