Spinal Protein Kinase M ζ Regulates α-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid Receptor Trafficking and Dendritic Spine Plasticity via Kalirin-7 in the Pathogenesis of Remifentanil-induced Postincisional Hyperalgesia in Rats

Conclusions Spinal protein kinase M ζ regulation of GluA1-containing AMPA receptor trafficking and spine morphologyvia Kalirin-7 overexpression is a fundamental pathogenesis of remifentanil-induced hyperalgesia in rats.
Source: Anesthesiology - Category: Anesthesiology Source Type: research