Gpr97 Exacerbates AKI by Mediating Sema3A Signaling

Conclusions Gpr97 is an important mediator of AKI, and pharmacologic targeting of Gpr97-mediated Sema3A signaling at multiple levels may provide a novel approach for the treatment of AKI.

Source: Journal of the American Society of Nephrology : JASN - April 30, 2018 Category: Urology & Nephrology Authors: Fang, W., Wang, Z., Li, Q., Wang, X., Zhang, Y., Sun, Y., Tang, W., Ma, C., Sun, J., Li, N., Yi, F. Tags: Basic Research Source Type: research