Role of upregulated miR-136-5p in lung adenocarcinoma: a study of 1242 samples utilizing bioinformatics analysis

Conclusion MiR-136-5p is overexpressed in lung adenocarcinoma and is involved in the molecular mechanism of lung adenocarcinoma via suppressing the expressions of downstream targets, especially claudin-18, sialophorin and syndecan 2 that participate in cell adhesion.
Source: Pathology Research and Practice - Category: Pathology Source Type: research