Inhibition of miR-186-5p contributes to high glucose-induced injury in AC16 cardiomyocytes.

In conclusion, these results suggest that the distribution of miR-186-5p contributes to HG-induced cytotoxicity and apoptosis in AC16 cardiomyocytes. PMID: 29399065 [PubMed]
Source: Experimental and Therapeutic Medicine - Category: General Medicine Tags: Exp Ther Med Source Type: research