Inhibition of miR-186-5p contributes to high glucose-induced injury in AC16 cardiomyocytes.
In conclusion, these results suggest that the distribution of miR-186-5p contributes to HG-induced cytotoxicity and apoptosis in AC16 cardiomyocytes.
PMID: 29399065 [PubMed]
Source: Experimental and Therapeutic Medicine - Category: General Medicine Tags: Exp Ther Med Source Type: research
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