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Dr. Helen Genova awarded grant for novel emotional processing study in multiple sclerosis

(Kessler Foundation) Helen Genova, Ph.D., Assistant Director of Neuropsychology&Neuroscience Research at Kessler Foundation, has been awarded a $44,000 grant from the National Multiple Sclerosis Society. The grant funds her research on emotional processing deficits in people with multiple sclerosis, an autoimmune degenerative disease of the central nervous system associated with significant behavioral and emotional sequelae.
Source: EurekAlert! - Medicine and Health - Category: International Medicine & Public Health Source Type: news

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Multiple sclerosis (MS) is an autoimmune and neurodegenerative disease of the central nervous system (CNS) with different types of disease courses (relapsing-remitting, secondary-progressive, primary progressive) that leads to physical as well as mental di...
Source: SafetyLit - Category: International Medicine & Public Health Tags: Ergonomics, Human Factors, Anthropometrics, Physiology Source Type: news
This article is protected by copyright. All rights reserved. PMID: 29331024 [PubMed - as supplied by publisher]
Source: Immunology - Category: Allergy & Immunology Authors: Tags: Immunology Source Type: research
Conclusions: The functions of IL-27 are currently unclear because it appears to affect different cells, cytokines, and signaling pathways differently. Further investigations are needed to clarify the role of IL-27 in autoimmune diseases. Hence, this article reviewed the multiple roles of IL-27 in autoimmune diseases and describe the influence of IL-27 on the immune system.
Source: Current Immunology Reviews - Category: Allergy & Immunology Source Type: research
This article is protected by copyright. All rights reserved.
Source: Annals of Neurology - Category: Neurology Authors: Tags: Research Article Source Type: research
Publication date: February 2018 Source:Brain, Behavior, and Immunity, Volume 68 Author(s): Fabrizia C. Guarnieri, Serena Bellani, Latefa Yekhlef, Andrea Bergamaschi, Annamaria Finardi, Riccardo Fesce, Davide Pozzi, Elena Monzani, Eugenio F. Fornasiero, Michela Matteoli, Gianvito Martino, Roberto Furlan, Stefano Taverna, Luca Muzio, Flavia Valtorta The classical view of multiple sclerosis (MS) pathogenesis states that inflammation-mediated demyelination is responsible for neuronal damage and loss. However, recent findings show that impairment of neuronal functions and demyelination can be independent events, suggesting the...
Source: Brain, Behavior, and Immunity - Category: Neurology Source Type: research
FINDINGSA study by UCLA researchers reveals the cellular basis for how the hormone estrogen protects against damage to the central nervous system in women with multiple sclerosis, or MS. The researchers found that estrogen treatment exerts positive effects on two types of cells during disease — immune cells in the brain as well as cells called oligodendrocytes.BACKGROUNDUCLADr. Rhonda VoskuhlMultiple sclerosis is a chronic autoimmune, neurodegenerative disease marked by visual impairment, weakness and sensory loss, as well as cognitive decline. These symptoms emerge when inflammatory immune cells destroy the myelin s...
Source: UCLA Newsroom: Health Sciences - Category: Universities & Medical Training Source Type: news
FINDINGSA UCLA study found that gene expression in specific cells and in specific regions of the body can provide a more precise, neuroprotective approach than traditional treatments for neurological diseases. Gene expression is the process by which genetic instructions are used to synthesize gene products, such as proteins, which go on to perform essential functions.For multiple sclerosis, specifically, increasing cholesterol synthesis gene expression in astrocytes of the spinal cord can be a pathway to repair nerves that affect walking.BACKGROUNDMultiple sclerosis is an autoimmune, neurodegenerative disease characterized...
Source: UCLA Newsroom: Health Sciences - Category: Universities & Medical Training Source Type: news
ppo M Abstract During multiple sclerosis (MS), a close link has been demonstrated to occur between inflammation and neuro-axonal degeneration, leading to the hypothesis that immune mechanisms may promote neurodegeneration, leading to irreversible disease progression. Energy deficits and inflammation-driven mitochondrial dysfunction seem to be involved in this process. In this work we investigated, by the use of striatal electrophysiological field-potential recordings, if the inflammatory process associated with experimental autoimmune encephalomyelitis (EAE) is able to influence neuronal vulnerability to the block...
Source: Neurobiology of Disease - Category: Neurology Authors: Tags: Neurobiol Dis Source Type: research
más G, Molnár G Abstract Degradation of myelin sheath is thought to be the cause of neurodegenerative diseases, such as multiple sclerosis (MS), but definitive agreement on mechanism of how myelin is lost is currently lacking. Autoimmune initiation of MS has been recently questioned by proposing that the immune response is a consequence of oligodendrocyte degeneration. To study the process of myelin breakdown, we induced demyelination with cuprizone and applied coherent anti-Stokes Raman scattering (CARS) microscopy, a non-destructive label-free method to image lipid structures in living tissue. We c...
Source: Brain Research Bulletin - Category: Neurology Authors: Tags: Brain Res Bull Source Type: research
ConclusionAlthough nearly half of the Dutch MS multiplex families reported an AID, no excess of AIDs was present in patients with MS from multiplex families or their first‐degree family members compared to the spouses.
Source: Acta Neurologica Scandinavica - Category: Neurology Authors: Tags: ORIGINAL ARTICLE Source Type: research
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