Analysis of temperature increase in swine gingiva after exposure to a Polywave ® LED light curing unit
Light emitting diode (LED) light curing units (LCU) have become one of the most important tools in the clinicians' routine. Although earlier LED LCU generations were considered “cool” lights because they generated less heat than did quartz-tungsten-halogen (QTH) lamps [1–5], later, more powerful LCU versions were capable of generating as much heat as QTH sources [4,6–8]. Most recently, a new generation of LED lamp has been introduced. Different from the previous ge nerations, these polywave® or “dual-peak” (multi-wave, multi-peak) LCUs emit light a broader wavelength range  to activate not only traditional photoinitiators, such as camphorquinone, but also alternative photoinitiators [10,11].
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We describe the clinical course of a 2-month-old infant who was evaluated for autosomal dominant Hyper IgE Syndrome based on eczema, periorbital cellulitis, skin abscesses, increased total IgE levels and blood eosinophilia. However, scabies and nasal colonization by Panton-Valentine Leucocidin-positive S. aureus were eventually diagnosed. After specific treatment, the child was asymptomatic.
This report describes the first Enterovirus D68 detections in acute flaccid paralysis cases occurring between December 2015 and March 2016 in Spain.
A 13-year-old male patient presented with a complaint of swelling of the left eye starting 3 days ago. Bullous lesion and purulent discharge were present on his left eyelids. Bacillus anthracis was shown in culture and diagnosis was confirmed. Oculocutaneous anthrax is a rare condition, but the diagnosis should be considered in patients with a painless necrotizing ulcer.
Post-malaria neurologic syndrome (PMNS) is a rare complication following a Plasmodium falciparum infection and its pathophysiology remains unclear. This is the first report of a pediatric PMNS following an infection acquired in Africa and the fourth description of pediatric PMNS overall. Neither intrathecal synthesis of Immunoglobin G nor specific P. falciparum antibodies were found in the cerebrospinal fluid.
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