Airway and Pulmonary β2-adrenergic Vasodilatory Function in Current-smokers and Never-smokers.

CONCLUSIONS: These results are consistent with a dysfunction within a β2-adrenergic signaling pathway mediating vasorelaxation in the airway circulation of current-smokers. The vasodilatory deficit in the airway but not the pulmonary circulation could be related to local differences in the impact of cigarette smoke on the vascular endothelium. PMID: 28012803 [PubMed - as supplied by publisher]

https://www.ncbi.nlm.nih.gov/pubmed/28012803?dopt=Abstract

Source: Chest - December 20, 2016 Category: Respiratory Medicine Authors: Hurwitz BE, Mendes ES, Schmid A, Parker M, Arana J, Gonzalez A, Wanner A Tags: Chest Source Type: research