Uppsala Consensus Statement on Environmental Contaminants and the Global Obesity Epidemic

Conclusions In conclusion, since there are now numerous animal and epidemiological studies indicating that environmental pollutants could contribute to the global obesity epidemic, there is an urgent need to reduce the burden of environmental contaminants so that obesity does not become the normal outlook in the future. The workshop attendees concluded that public health efforts should focus on the importance of early obesity prevention by means of reducing chemical exposures, rather than only treating the established disease. Just as a bad start can last a lifetime and beyond, a good start can last a lifetime as well. References Agay-Shay K, Martinez D, Valvi D, Garcia-Esteban R, Basagana X, Robinson O, et al. 2015. Exposure to endocrine-disrupting chemicals during pregnancy and weight at 7 years of age: a multi-pollutant approach. Environ Health Perspect 123(10):1030–1037, doi: 10.1289/ehp.1409049. Alonso-Magdalena P, Quesada I, Nadal A. 2011. Endocrine disruptors in the etiology of type 2 diabetes mellitus. Nat Rev Endocrinol 7(6):346–353, doi: 10.1038/nrendo.2011.56. Angle BM, Do RP, Ponzi D, Stahlhut RW, Drury BE, Nagel SC, et al. 2013. Metabolic disruption in male mice due to fetal exposure to low but not high doses of bisphenol A (BPA): evidence for effects on body weight, food intake, adipocytes, leptin, adiponectin, insulin and glucose regulation. Reprod Toxicol 42(12):256–268, doi: 10.1016/j.reprotox.2013.07.017. Brown RE, Sharma AM, Ardern CI, Mirdamadi P, M...
Source: EHP Research - Category: Environmental Health Authors: Tags: Brief Communication May 2016 Source Type: research