E-Cigarettes: Hooking a New Generation
Recently a father contacted CEH with concerns about his 16-year-old son's health. The teenager had purchased nicotine-laced e-liquids from a website that had no age verification or nicotine warnings. The packaging on the e-liquid also had no warning labels, despite the known health consequences of exposure to nicotine. Angry and frustrated at the lack of rules on e-cigarettes, the dad told us, "If you can help get them to do the right thing, that would be awesome." Parents around the country are now realizing that they need to learn more about e-cigarettes and their children's health. According to recent research by the Centers for Disease Control, the e-cigarette industry aggressively targets teens in their advertising, with nearly 70 percent of middle and high school students exposed to e-cigarette ads. As the tobacco industry knows from a century of experience, their advertising works: E-cigarette use is skyrocketing among teenagers. "Vaping" proponents say that e-cigarettes are primarily used by smokers as a cessation aid, but according to CDC chief Dr. Tom Friedman, the tobacco industry has another reason for the spate of ad dollars targeted at our kids. As he told the Los Angeles Times, "The same advertising tactics the tobacco industry used years ago to get kids addicted to nicotine are now being used to entice a new generation of young people to use e-cigarettes." Meanwhile the evidence of health risks from e-cigarettes continues to moun...
Officially it ’s a ‘unique holiday hotel for cancer patients and those with life-threatening illnesses’, according to the sign outside the Twenties green-shuttered building in Bournemouth.
Sarah Brookes, 41, a mother of two from Bradford, West Yorkshire, would like commercial sunbeds to be banned outright.
Publication date: August 2018Source: Biochimica et Biophysica Acta (BBA) - Reviews on Cancer, Volume 1870, Issue 1Author(s): Chi Van Dang
Publication date: August 2018Source: Biochimica et Biophysica Acta (BBA) - Reviews on Cancer, Volume 1870, Issue 1Author(s):
The steep increase in thyroid cancer diagnosis has not been mirrored by increasing mortality rates and is likely due to widespread overdiagnosis of small neoplasms.
The presence of HER3 expression and a high HER2/CEP17 ratio distinguish a group of invasive HER2-positive ductal breast cancer patients who have a worse prognosis.
Publication date: 13 August 2018Source: Cancer Cell, Volume 34, Issue 2Author(s): Manisha Rao, Scott PowersIn this issue of Cancer Cell, Menghi et al. show that tandem duplications (TDs) are a common genetic feature of gynecological and triple-negative breast cancers. They also provide evidence that these TDs generate genetic alterations driving these cancers, which, if substantiated, will fundamentally change our understanding of their oncogenesis.
Publication date: 13 August 2018Source: Cancer Cell, Volume 34, Issue 2Author(s): Carolyn Hurst, Jonathan Rosenberg, Margaret KnowlesBladder cancers, the majority of which are urothelial carcinoma, are the most common urinary tract cancers. There are two major disease types, muscle-invasive and non-muscle-invasive, with distinct pathogenesis pathways, molecular features, and clinical outcomes. Recent genomic and transcriptomic data have significantly improved tumor sub-classification and prognostication for both patient groups and are informing improved approaches to treatment of muscle-invasive disease. To view this SnapS...
Publication date: 13 August 2018Source: Cancer Cell, Volume 34, Issue 2Author(s): Gururaj Shivange, Karol Urbanek, Piotr Przanowski, Justin S.A. Perry, James Jones, Robert Haggart, Christina Kostka, Tejal Patki, Edward Stelow, Yuliya Petrova, Danielle Llaneza, Marty Mayo, Kodi S. Ravichandran, Charles N. Landen, Sanchita Bhatnagar, Jogender Tushir-SinghSummaryTherapeutic antibodies targeting ovarian cancer (OvCa)-enriched receptors have largely been disappointing due to limited tumor-specific antibody-dependent cellular cytotoxicity. Here we report a symbiotic approach that is highly selective and superior compared with in...
Publication date: 13 August 2018Source: Cancer Cell, Volume 34, Issue 2Author(s): Ramona Schulz-Heddergott, Nadine Stark, Shelley J. Edmunds, Jinyu Li, Lena-Christin Conradi, Hanibal Bohnenberger, Fatih Ceteci, Florian R. Greten, Matthias Dobbelstein, Ute M. MollSummaryOver half of colorectal cancers (CRCs) harbor TP53 missense mutations (mutp53). We show that the most common mutp53 allele R248Q (p53Q) exerts gain of function (GOF) and creates tumor dependence in mouse CRC models. mutp53 protein binds Stat3 and enhances activating Stat3 phosphorylation by displacing the phosphatase SHP2. Ablation of the p53Q allele suppres...
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