Lack of Galectin-3 Prevents Cardiac Fibrosis and Effective Immune Responses in a Murine Model of Trypanosoma cruzi Infection

Conclusions. These results suggest that galectin-3 is strongly involved in Chagas disease, not only in the immune response against T. cruzi, but also in mediating cardiac tissue damage.
Source: The Journal of Infectious Diseases - Category: Infectious Diseases Authors: Tags: PATHOGENESIS AND HOST RESPONSE Source Type: research