Atypical chemokine receptor 1 deficiency reduces atherogenesis in ApoE-knockout mice

Conclusions Ackr1 deficiency appears to be protective in the ApoE knockout model of atherogenesis, but it is associated with only modest changes in cytokine and chemokine expression as well as T-cell subset frequency and inflammatory macrophage content.
Source: Cardiovascular Research - Category: Cardiology Authors: Tags: Vascular biology Source Type: research