Targeting Glia to Treat Chronic Pain: Moving from Concept to Clinical Trials

Dr. Watkins' research is focused on how to control chronic pain and increase the effectiveness of analgesics while minimizing their side effects. When non-neuronal cells known as glia are activated, they can create persistent, amplified neuroinflammation, thereby promoting the transition from acute to chronic pain. Her work has shown that glial activation can also compromise the ability of opioids to suppress chronic pain, contributing to the development of drug tolerance and dependence. For more information go to http://www.nidcr.nih.gov/NewsAndFeatures/Announcements/Kreshover-2015.htmAir date: 5/4/2015 2:00:00 PM
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